Last edited by Nikom
Thursday, April 23, 2020 | History

8 edition of Apoptosis and Autoimmunity found in the catalog.

Apoptosis and Autoimmunity

From Mechanisms to Treatments

by

  • 228 Want to read
  • 8 Currently reading

Published by Wiley-VCH .
Written in English

    Subjects:
  • Allergies,
  • Medical,
  • Autoimmunity,
  • Medical / Nursing,
  • Life Sciences - Cytology,
  • Immunology,
  • Medical / Immunology,
  • Autoimmune diseases,
  • Apoptosis

  • Edition Notes

    ContributionsJoachim R. Kalden (Editor), Martin Herrmann (Editor)
    The Physical Object
    FormatHardcover
    Number of Pages392
    ID Numbers
    Open LibraryOL9052446M
    ISBN 103527304428
    ISBN 109783527304424

    Elkon K (ed): Apoptosis and Its Relevance to Autoimmunity. Curr Dir Autoimmun. Basel, Karger, , vol 9, pp 1–17 Death Receptor Signaling and Its Function in the Immune System Stefanie C. Fas, Benedikt Fritzsching, Elisabeth Suri-Payer, Peter H. Krammer Tumor Immunology Program, Division of Immunogenetics. How was apoptosis discovered? The term apoptosis was coined by John Kerr, Andrew Wyllie and A.R. Currie in The molecular basis of apoptosis was elucidated for the first time by the studies in a nematode Caenorhabditis worm C. elegans constantly maintain their cell number in its embryonic and adult stages. During the embryonic development, the worm .


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Apoptosis and Autoimmunity Download PDF EPUB FB2

This is the first comprehensive book about the relationship between apoptosis and autoimmune diseases. It offers a unique up-to-date overview on research results on the defective execution of apoptosis and the incomplete clearance of apoptotic cells.

The molecular and cellular mechanisms involved are described in detail. About this Book This is the first comprehensive book about the relationship between apoptosis and autoimmune diseases. It offers a unique up-to-date overview on research results on the defective execution of apoptosis and the incomplete clearance of apoptotic cells.

Part 1. General feature of apoptosis. Apoptosis and autoimmunity / Keith Elkon --Caspase knockouts: matters of life and death / Saquib Lakhani, Binfeng Lu, and Richard A.

Flavell --Part 2. Clearance of apoptotic cells. The largely incomplete concordance rates of autoimmune diseases in monozygotic twins strongly support other complementary mechanisms involved in gene regulation ultimately causing overt autoimmunity, and it is becoming increasingly evident that epigenetic deregulation contributes to the emergence and/or the progression of disorders that include.

This is the first comprehensive book about the relationship between apoptosis and autoimmune diseases. It offers a unique up-to-date overview on research results on the defective execution of apoptosis and the incomplete clearance of apoptotic cells. The molecular and cellular mechanisms involved are described in detail.

The cells of a multicellular organism are members of a highly organized community. The number of cells in this community is tightly regulated—not simply by controlling the rate of cell division, but also by controlling the rate of cell death. If cells are no longer needed, they commit suicide by activating an intracellular death program.

This process is therefore called programmed cell Cited by: Main Apoptosis and Autoimmunity: From Mechanisms to Treatments. Apoptosis and Autoimmunity: From Mechanisms to Treatments Joachim R. Kalden, Martin Herrmann. This is the first comprehensive book about the relationship between apoptosis and autoimmune diseases.

It offers a unique up-to-date overview on research results on the defective execution. Apoptosis and its relevance to autoimmunity. Basel ; New York: Karger, © (OCoLC) Online version: Apoptosis and its relevance to autoimmunity.

Basel ; New York: Karger, © (OCoLC) Material Type: Internet resource: Document Type: Book, Internet Resource: All Authors / Contributors: Keith B Elkon. In Medical Cell Biology (Third Edition), SUMMARY. Apoptosis is a morphologically and biochemically distinct form of programmed cell death that plays an essential role during embryologic development, after birth, and during adulthood.

However, deregulation of apoptosis is involved in the pathogenesis of a variety of human diseases. Since the late s, the core. The process of programmed cell death or apoptosis was already noted in by Vogt [1], but it was not until the more recent studies of Kerr et al.

[2] that an explosion of interest in apoptosis research occurred. Genetic, biochemical and cellular analysis in certain mammals, in the nematode Caenorhabditis elegans and in the fruitfly Drosophila melanogaster Cited by: Apoptosis and autoimmunity. This scientific book includes a glossary of terms, an updated selective reference list (of more than titles), and a large summary (of 12 pages) written in.

SERPINB3, apoptosis and autoimmunity Article Literature Review (PDF Available) in Autoimmunity reviews 9(2) April with Reads How we measure 'reads'. The book offers authoritative discussions on the role of apoptosis in the deletion of self-reactive lymphocytes, prevention of autoimmunity, termination of the immune response, death of T cells infected by the HIV virus, neoplasia, and the homeostatic regulation of the hematopoietic system.

Abstract. The thyroid is the most common target for organ-specific autoimmune destruction in humans. InHashimoto described 4 cases of goiter with diffuse lymphocyte infiltration, eosinophilic changes of follicular cells, and various degrees of atrophy and fibrosis (1).Cited by: 2.

Download Apoptosis and Its Relevance to Autoimmunity - book pdf free download link or read online here in PDF. Read online Apoptosis and Its Relevance to Autoimmunity - book pdf free download link book now.

All books are in clear copy here, and all files are secure so don't worry about it. The other track is a general susceptibility to autoimmunity through genes that influence tolerance, apoptosis, or inflammatory responses.

These genes explain the well-known tendency for autoimmunity to run in families, with multiple and variable expressions of disease in Cited by: 4. Tamar Aprahamian (March 9th ). Autoimmunity, Atherosclerosis and Apoptotic Cell Clearance, Inflammation, Chronic Diseases and Cancer - Cell and Molecular Biology, Immunology and Clinical Bases, Mahin Khatami, IntechOpen, DOI: / Available from:Cited by: 1.

Autoimmunity is the system of immune responses of an organism against its own healthy cells and tissues. Any disease that results from such an aberrant immune response is termed an "autoimmune disease".Prominent examples include celiac disease, post-infectious IBS, diabetes mellitus type 1, Henloch Scholein Pupura (HSP) sarcoidosis, systemic lupus erythematosus Specialty: Immunology.

Elkon KB, Oberst A. Apoptosis and inflammatory forms of cell death. In: Dubois' Lupus Erythematosus and Related Syndromes, 9th ed, Wallace DJ, Hahn BH (Eds), Elsevier, p Dwivedi N, Radic M. Citrullination of autoantigens implicates NETosis in the induction of autoimmunity.

Ann Rheum Dis ; Linkermann A, Green DR. Necroptosis. Apoptosis is a normal part of development and is required during development, creation of the central nervous system, degeneration of the tadpole tail during metamorphosis, and the loss of certain appendages during the larval to pupal metamorphosis in holometabolous insects.

Apoptosis is a major aspect of development and by: 1. General description This is the first comprehensive book about the relationship between apoptosis and autoimmune diseases.

It offers a unique up-to-date overview on research results on the defective execution of apoptosis and the incomplete clearance of apoptotic cells. While apoptosis is an important process for metazoan cells, defective apoptosis can have a number of negative impacts on the organism (e.g.

degenerative diseases and autoimmunity). Based on research studies, development and progression of cancer have been shown to be one of these outcomes. Cell Cannibalism, Apoptosis and Autoimmunity Presenter: Dr Manjunatha T M Slideshare uses cookies to improve functionality and performance, and to provide you with relevant advertising.

If you continue browsing the site, you. Understand the concepts of autoimmunity and disease Know the features of major autoimmune diseases Know the theories on etiology of autoimmune disease: TOLERANCE.

Introduction. Tolerance refers to the specific immunological non-reactivity to an antigen resulting from a previous exposure to the same antigen. While the most important form of. The term apoptosis (a-po-toe-sis) was first used in a now-classic paper by Kerr, Wyllie, and Currie in to describe a morphologically distinct form of cell death, although certain components of the apoptosis concept had been explicitly described many years previously (Kerr et al., ; Paweletz, ; Kerr, ).Our understanding of the mechanisms involved Cited by: Apoptosis (from Ancient Greek ἀπόπτωσις, apóptōsis, "falling off") is a form of programmed cell death that occurs in multicellular organisms.

Biochemical events lead to characteristic cell changes and changes include blebbing, cell shrinkage, nuclear fragmentation, chromatin condensation, chromosomal DNA fragmentation, and global [vague] mRNA : D   Systems Biology of Apoptosis summarizes all current achievements in this emerging field. Apoptosis is a process common to all multicellular organisms.

Apoptosis leads to the elimination of cells via a complex but highly defined cellular programme. Defects in the regulation of apoptosis result in serious diseases such as cancer, autoimmunity, AIDS and. of ‘autoimmunity’. More recently, the relationship between autoimmunity and apoptosis has been the focus of much research activity.

Apoptosis as a genetically predetermined pro-cess is not only a vital mechanism sustaining homeostasis in the regulation of im-mune reactivity, but, in addition to being an important factor in general cell phy. Since publication of the 4th Edition of The Autoimmune Diseases inthe understanding of the immune mechanisms underlying autoimmunity and autoimmune disease has significantly deepened and broadened.

This fully revised 5th Edition incorporates new material and combines common themes underlying inductive and effector mechanisms and therapies Book Edition: 5. Apoptosis and Autoimmunity read book. Apoptosis and Autoimmunity PDF. From Mechanisms to Treatments. Authors: Joachim Kalden R., Martin Herrmann.

rate. Reviews. Read preview. Save. Add to cart. Buy and read for $ WELCOME, LET THE FUN BEGIN. Get e-Books "B Cell Biology In Autoimmunity" on Pdf, ePub, Tuebl, Mobi and Audiobook for are more than 1 Million Books that have been enjoyed by people from all over the world.

Always update books hourly, if not looking, search in the book search column. Enjoy % FREE. The Role of Apoptosis in Thyroid Autoimmunity Su He Wang and James R. Baker, Jr. There is increasing evidence showing that apoptosis plays a role in the development of the autoimmune thyroid diseases—Hashimoto’s (lymphocytic) thyroiditis (HT) and Graves’ disease (GD).

The immune pathogenesis of HT. • Autoimmunity involves a misdirection of the body’s immune system against its own tissues, causing a large number of diseases. • More than 80 autoimmune diseases have so far been identified: some affect only one tissue or organ, while others are ‘systemic’ (affection multiple sites of.

Apoptosis and autoimmunity are two phenomena, which share essential similarities in destroying cells. However, apoptosis is a normal process, while autoimmunity is not. Apoptosis could, at least in some conditions, cause autoimmunity. The failure to appropriately achieve programmed cell death and to clear apoptotic cell fragments is.

Keywords:Autoimmunity, apoptosis, autoantigen, autoantibody, adaptive immune response, innate immune system, tolerance. Abstract: Acquisition of a complex immune system during evolution provided organisms with the most effective defense mechanism against “foreign” or “non-self” invaders.

This efficient protection against pathogens Cited by: Autoimmunity and chronic diseases autoimmune, autoimmunity, chronic, fibromyalgia, inflammation, lupus, multiple-sclerosis, polymyalgia.

13 likes Like. Lists are re-scored approximately every 5 minutes. People Who Voted On This List (20) Incorrect Book The list contains an incorrect book (please specify the title of the book.

Iodine and Apoptosis: Iodine is needed by every cell in the body, but tends to concentrate in areas where there is frequent cell regeneration (which involves apoptosis).This includes nasal passages and mucus cells of the stomach lining, where cell regeneration takes place very days.

If there is insufficient iodine in the body to perform this critical function. Since publication of the 4th Edition of The Autoimmune Diseases inthe understanding of the immune mechanisms underlying autoimmunity and autoimmune disease has significantly deepened and broadened.

This fully revised 5th Edition incorporates new material and combines common themes underlying inductive and effector mechanisms and therapies. Moved Permanently. openresty. Autoimmunity It is generally believed that autoimmunity is triggered by the development or activation of CD4 helper T cells that react with a specific autoantigen.

Based on various evidence, it is now proposed that a specific antigenic stimulus is the first trigger of autoimmunity. This is called the “single initiating antigen hypoth-esis”. Autoimmunity and Apoptosis – Therapeutic Implications Iran Rashedi1,4, Soumya Panigrahi2,4, Peyman Ezzati1,4, Saeid Ghavami1,4 and Marek Los*,1,3,4,5 1Department of Biochemistry and Medical Genetics, 2Department of Physiology, 3 Department of Human Anatomy and.

The new book summarizes the international scientific research on the possible treatment of autoimmunity with BCG or TNF induction, and provides a rationale for the use of BCG at the forefront of. Saleh Altuwaijri, Kuang-Hsiang Chuang, Kuo-Pao Lai, Jiann-Jyh Lai, Hung-Yun Lin, Faith M.

Young, Andrea Bottaro, Meng-Yin Tsai, Wei-Ping Zeng, Hong-Chiang Chang, Shuyuan Yeh, Chawnshang Chang, Susceptibility to Autoimmunity and B Cell Resistance to Apoptosis in Mice Lacking Androgen Receptor in B Cells, Molecular Endocrinology, Volume Cited by: